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Proteomic analysis of Nrf2 deficient transgenic mice reveals cellular defence and lipid metabolism as primary Nrf2-dependent pathways in the liver

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20 To circumvent these problems, we generated mice selectively lacking IR in chondrocytes through cross-breeding of the mouse strain carrying the IR-floxed allele 21 and the mouse strain carrying Cre recombinase under the control of collagen type II promoter

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Their value is highest when resistance training, protein intake, calorie control, sleep, and recovery habits are already in place